Titlebook: Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity; Santiago Grisolía,Vicente Felipo,María-Dolores Miñ Book 1990 The Editor(s) (if a

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书目名称Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity影响因子(影响力)

书目名称Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity影响因子(影响力)学科排名

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书目名称Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity读者反馈学科排名

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0065-2598 Overview: 978-1-4684-5828-2978-1-4684-5826-8Series ISSN 0065-2598 Series E-ISSN 2214-8019

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https://doi.org/10.1007/978-1-4614-5419-9multifactorial syndrome and that, in general, in its chronic recurrent presentation or in acutely precipitated coma, the episode of encephalopathy is reversible unless the precipitating factor per se carries a high degree of mortality (i.e. massive gastrointestinal bleeding, fulminant viral infection, etc) (1).

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Treatment of Portal Systemic Encephalopathy: The Old and New Treatmentsmultifactorial syndrome and that, in general, in its chronic recurrent presentation or in acutely precipitated coma, the episode of encephalopathy is reversible unless the precipitating factor per se carries a high degree of mortality (i.e. massive gastrointestinal bleeding, fulminant viral infection, etc) (1).

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Sexualität und Recht: eine Einführung systemic shunting (1, 2, 3). Whether the development of encephalopathy is caused by diversion of blood past the liver, decreased liver function, or both, is unknown. The symptoms of hepatic encephalopathy caused by portacaval shunting (pcs) or chronic liver dysfunction in man range from very subtle

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Eheschließungsfreiheit im Kampf der Kulturens ammonia is neurotoxic and its concentration must be kept low. Ammonia generated in most extrahepatic tissues is detoxified by incorporation into glutamine (amide). This glutamine may be used in a number of biosynthetic reactions (e.g. in pyrimidine synthesis). Alternatively, as a means of maintain

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https://doi.org/10.1007/978-3-663-08200-2n integrated picture of all of the available facts and events regarding the pathogenesis of this condition has yet to emerge. Many studies to date have largely ignored certain fundamental cellular changes which occur in this condition. In this article, the involvement of astrocytes in HE will be rev

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Titlebook: Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity; Santiago Grisolía,Vicente Felipo,María-Dolores Miñ Book 1990 The Editor(s) (if a

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